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Folate Metabolism Interferes with Plant Immunity through 1C Methionine Synthase-Directed Genome-wide DNA Methylation Enhancement

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Folate Metabolism Interferes with Plant Immunity through 1C Methionine Synthase-Directed Genome-wide DNA Methylation Enhancement

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González, B.; Vera Vera, P. (2019). Folate Metabolism Interferes with Plant Immunity through 1C Methionine Synthase-Directed Genome-wide DNA Methylation Enhancement. Molecular Plant. 12(9):1227-1242. https://doi.org/10.1016/j.molp.2019.04.013

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Título: Folate Metabolism Interferes with Plant Immunity through 1C Methionine Synthase-Directed Genome-wide DNA Methylation Enhancement
Autor: González, Beatriz Vera Vera, Pablo
Entidad UPV: Universitat Politècnica de València. Instituto Universitario Mixto de Biología Molecular y Celular de Plantas - Institut Universitari Mixt de Biologia Molecular i Cel·lular de Plantes
Fecha difusión:
Resumen:
[EN] Plants rely on primary metabolism for flexible adaptation to environmental changes. Here, through a combination of chemical genetics and forward genetic studies in Arabidopsis plants, we identified that the essential ...[+]
Palabras clave: Folate pathway , Plant immunity , Methionine , DNA methylation
Derechos de uso: Reconocimiento (by)
Fuente:
Molecular Plant. (issn: 1674-2052 )
DOI: 10.1016/j.molp.2019.04.013
Editorial:
Elsevier
Versión del editor: https://doi.org/10.1016/j.molp.2019.04.013
Código del Proyecto:
info:eu-repo/grantAgreement/MINECO//BFU2015-68199-R /ES/SOBRE LOS MECANISMOS DE SUSCEPTIBILIDAD A PATOGENOS EN ARABIDOPSIS/
info:eu-repo/grantAgreement/AEI/Plan Estatal de Investigación Científica y Técnica y de Innovación 2017-2020/RTI2018-098501-B-I00/ES/NUEVOS MEDIADORES DE LA ACTIVACION DE MECANISMOS DE RESISTENCIA Y DE FACTORES SUSCEPTIBILIDAD DE LA PLANTA A MICROORGANISMOS PATOGENOS/
Agradecimientos:
We acknowledge V. Flors for the determination of SA level, S. Tarraga for the 2D-DIGE analysis and selection of spots proteins, and the SCSIE_University of Valencia Proteomics Unit for MS-MALDI-TOF/TOF and/or LC-MS/MS ...[+]
Tipo: Artículo

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