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Inactivation of PYR/PYL/RCAR ABA receptors by tyrosine nitration may enable rapid inhibition of ABA signaling by nitric oxide in plants

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Inactivation of PYR/PYL/RCAR ABA receptors by tyrosine nitration may enable rapid inhibition of ABA signaling by nitric oxide in plants

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Castillo López Del Toro, MC.; Lozano Juste, J.; Gonzalez Guzman, M.; Rodriguez, L.; Rodríguez Egea, PL.; Leon Ramos, J. (2015). Inactivation of PYR/PYL/RCAR ABA receptors by tyrosine nitration may enable rapid inhibition of ABA signaling by nitric oxide in plants. Science Signaling. 8(392):1-10. doi:10.1126/scisignal.aaa7981

Por favor, use este identificador para citar o enlazar este ítem: http://hdl.handle.net/10251/85131

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Title: Inactivation of PYR/PYL/RCAR ABA receptors by tyrosine nitration may enable rapid inhibition of ABA signaling by nitric oxide in plants
Author: Castillo López del Toro, Mª Cruz Lozano Juste, Jorge González Guzmán, Miguel Rodriguez, Lesia Rodríguez Egea, Pedro Luís Leon Ramos, Jose
UPV Unit: Universitat Politècnica de València. Instituto Universitario Mixto de Biología Molecular y Celular de Plantas - Institut Universitari Mixt de Biologia Molecular i Cel·lular de Plantes
Universitat Politècnica de València. Departamento de Biotecnología - Departament de Biotecnologia
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Abstract:
[EN] Abscisic acid (ABA) is a phytohormone that inhibits growth and enhances adaptation to stress in plants. ABA perception and signaling rely on its binding to receptors of the pyrabactin resistance1/PYR1-like/regulatory ...[+]
Subjects: 2C protein phosphatases , Abscisic acid receptors , Arabidopsis thaliana , Drought resistance , Stomatal closure , Plasma membrane , Seed dormancy , In vivo , Peroxynitrite , Nitrosylation
Copyrigths: Cerrado
Source:
Science Signaling. (issn: 1937-9145 ) (eissn: 1937-9145 )
DOI: 10.1126/scisignal.aaa7981
Publisher:
American Association for the Advancement of Science
Publisher version: http://doi.org/10.1126/scisignal.aaa7981
Thanks:
This work was supported by the Ministry of Science and Innovation (MICINN) (Spain) grants BIO2008-00839 and BIO2011-27526 (to J.L.) and BIO2011-23446 (to P.L.R.).
Type: Artículo

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