- -

Mechanistic investigation of Ca2+ alternans in human heart failure and its modulation by fibroblasts

RiuNet: Institutional repository of the Polithecnic University of Valencia

Share/Send to

Cited by

Statistics

Mechanistic investigation of Ca2+ alternans in human heart failure and its modulation by fibroblasts

Show full item record

Mora-Fenoll, MT.; Gomez, JF.; Morley, G.; Ferrero De Loma-Osorio, JM.; Trenor Gomis, BA. (2019). Mechanistic investigation of Ca2+ alternans in human heart failure and its modulation by fibroblasts. PLoS ONE. 14(6):1-19. https://doi.org/10.1371/journal.pone.0217993

Por favor, use este identificador para citar o enlazar este ítem: http://hdl.handle.net/10251/146434

Files in this item

Item Metadata

Title: Mechanistic investigation of Ca2+ alternans in human heart failure and its modulation by fibroblasts
Author: Mora-Fenoll, María Teresa Gomez, Juan F. Morley, Gregory Ferrero De Loma-Osorio, José María Trenor Gomis, Beatriz Ana
UPV Unit: Universitat Politècnica de València. Departamento de Ingeniería Electrónica - Departament d'Enginyeria Electrònica
Issued date:
Abstract:
[EN] Background Heart failure (HF) is characterized, among other factors, by a progressive loss of contractile function and by the formation of an arrhythmogenic substrate, both aspects partially related to intracellular ...[+]
Subjects: Sarcoplasmic-Reticulum Ca2+ , Action-Potential dynamics , Electrical alternans , Cardiac alternans , Ventricular myocytes , Cellular alternans , Conduction , Model , Repolarization , Tissue
Copyrigths: Reconocimiento (by)
Source:
PLoS ONE. (issn: 1932-6203 )
DOI: 10.1371/journal.pone.0217993
Publisher:
Public Library of Science
Publisher version: https://doi.org/10.1371/journal.pone.0217993
Project ID:
FEDER/DPI2016-75799-R
UPV/PAID-01-17
AEI/DPI2016-75799-R
Thanks:
This work was partially supported by the Plan Estatal de Investigación Científica y Técnica y de Innovación 2013 2016" from the Ministerio de Economía, Industria y Competitividad of Spain and Fondo Europeo de Desarrollo ...[+]
Type: Artículo

This item appears in the following Collection(s)

Show full item record